The zebrafish resumed active, coordinated swimming and responded significantly better to light and movement, reflecting restored vision. Retinal architecture was structurally restored, including: ...

The study utilized the Sen57wrn-/-ND6-/+ zebrafish model, which combines genetic mutations associated with premature aging (WRN), mitochondrial dysfunction (ND6), and chronic senescence (Sen57).

The zebrafish resumed active, coordinated swimming and responded significantly better to light and movement, reflecting restored vision. Retinal architecture was structurally restored, including: ...

The study utilized the Sen57wrn-/-ND6-/+ zebrafish model, which combines genetic mutations associated with premature aging (WRN), mitochondrial dysfunction (ND6), and chronic senescence (Sen57). These ...

These new cells effectively replace the damaged cells with functional retinal neurons, a pretty neat trick. Sadly, more than 375 million years of evolution separate humans and zebrafish, so ...

Inherited retinal diseases are rare eye conditions that lead to progressive vision loss due to the degeneration of cells in the retina, the light-sensitive layer at the back of the eye.

In experiments with zebrafish, the team has now managed to fill this knowledge gap. Their findings reveal that spliceosome subunits U4, U5 and U6 are normally stabilized as a complex by a protein ...

“We were measuring tissue stiffness in the zebrafish retina, and realized that it depended on the packing of nuclei. This was totally unexpected because tissue mechanics is believed to depend on cell ...

He explains, "We were measuring tissue stiffness in the zebrafish retina, and realized that it depended on the packing of nuclei.